AIUI the mechanism for this is via inhibiting JAK/STAT
IMO this is about reducing the effect of IL-10 and therefore increasing NF kappa B, thereby increasing the level of the citrate/tricarboxylate transport protein and as a consequence increasing acetyl-CoA levels in the nucleus.
There are other Jak inhibitors and there is plenty of material around (see link below). However, I personally am concerned about impacting on the NF kappa B pathway. It has its role when it needs to be increased and/or reduced.