Cause of death has been determined:
Los Angeles County Department of Public Health that indicate Curry died of coronary artery disease. The public health department’s document also listed a history of stroke and kidney cancer as other significant conditions that contributed to his death, but did not result in the underlying cause of Curry’s death.
He did also have a major stroke in 2012 which left him disabled.
Maybe he didn’t like the nickname “Tiny” and decided to do something about it?
Apparently nobody wants to be tiny in any way - that’s why the other guy died in a gym, and btw. steroids are also taken in the hope of not being tiny.
Clearly avoiding being tiny can be lethal. But it goes the other way too. Being tiny is associated with longevity interspecies - smaller dogs live longer. Ames dwarf mice live much longer than regular size mice. You can live a long life as a sexually frustrated tiny person, and even diminish yourself further by chopping off the family jewels - eunuchs live longer (spayed/neutered pets live longer and healthier lives too).
Who wants to be tiny? Nobody, that’s who. Even folks on this site do all they can to become bigger and more sexually prolific. Give me size or give me death - OK, death it is, thank you. But you leave a bigger corpse behind, and that counts for something too.
I come from a very tall family, and apparently my mom ran out of those cells by the time I came around 
I look comical in family photos !
(I was 5’5” before I started shrinking, so I’m only relatively short compared to them!)
I try to tell myself, ok, this isn’t all bad because I might live longer!
Tall people - more cancer, less heart disease, short people the other way around. Ergo, you should pay attention to your CV health 
…
One major celebrity death not mentioned yet is Hayden Panettiere, aged 36. Likely cause seems to be drug overdose.
What’s crazy is, he was already married to some model, and their instagram seemed to be nothing but simply showing off how wealthy they were with their luxury lifestyle, nice holidays, London penthouse etc etc. So really, if a dude is still obsessed with his size down there, that’s some sort of mental disorder, body dysmorphia. Dude is already married - so who cares!
How HA could cause PE? Its impossible. Probably a coincidence.
I agree so I asked Chatgpt:
How HA enlargement works
The material is a thick, cross-linked hyaluronic-acid gel—essentially a clear, water-holding jelly. It is injected in a layer beneath the shaft skin but outside the erectile bodies.
It simply occupies space:
- The gel forms a sleeve or padding around the shaft.
- Because HA holds water, the injected volume may swell somewhat after placement.
- This increases circumference in both the flaccid and erect states.
- Any apparent increase in length is mostly because the heavier, bulkier penis retracts less when flaccid.
It does not grow new erectile tissue, enlarge the internal chambers, or permanently change the penis. The body gradually breaks the HA down, although repeated injections can leave fibrosis, lumps or irregularities.
Ubiribo reportedly received 40 mL—about eight teaspoons—in one session, reportedly his fifth treatment since 2024. That is an extraordinary amount compared with the 1–3 mL commonly used during many facial-filler procedures. The clinic claims its doctor recommended splitting the treatment into two sessions, although the investigation has not yet established whether malpractice occurred. People’s account of the inquest and investigation
How it could reach his lungs
The gel was supposed to remain in the tissue. It could not simply soak through the body in bulk. It almost certainly entered a vein directly, either because:
- the needle or cannula was inside or damaged a penile vein;
- filler was forced into an injured vessel under injection pressure; or
- it entered the extremely vascular erectile tissues or venous channels.
From there the route is direct:
penile veins → pelvic veins → vena cava → right side of heart → pulmonary arteries
The lungs are the first fine vascular “filter” encountered by anything injected into a vein. The HA gel can travel as numerous globs or strands and lodge throughout vessels of different sizes.
This exact phenomenon had already been documented before Ubiribo’s death. In a published fatal penile-enlargement case, autopsy found jelly-like HA around the penis and the same material within numerous lung vessels. The authors called it non-thrombotic pulmonary embolism—the emboli were filler rather than ordinary blood clots. 2025 forensic case report in PubMed
Did all 40 mL reach his lungs?
Probably not, and the public inquest reports do not tell us how much did. But even part of 40 mL entering the circulation would be an enormous embolic load. It could scatter through thousands of small pulmonary vessels and produce several effects simultaneously:
- Mechanical obstruction: Gel physically blocks pulmonary blood flow.
- Acute pulmonary hypertension: The right ventricle suddenly has to pump against a nearly closed vascular bed.
- Right-heart failure: The right ventricle dilates and fails; very little blood reaches the left heart.
- Inflammation and coagulation: Foreign gel can injure vessel linings and cause platelets and fibrin to accumulate around it, adding real blood clots to the filler obstruction.
- Severe oxygen failure and cardiac arrest.
That explains the rapid chest pain, fainting and collapse reported shortly after his procedure. His postmortem reportedly found material in the lungs consistent with the injected filler, which strongly favors direct filler embolization rather than a coincidental leg clot. HA-related pulmonary complications are exceptionally rare but documented, including fatal cases. 2025 medical review
One confusing point is terminology: newspapers call every pulmonary embolism a “blood clot.” In this case, the more accurate description appears to be a pulmonary embolism made principally of HA filler, possibly with secondary clotting around it.
The HA molecule itself is not especially poisonous—it naturally exists in our bodies and is used in joints and eyes. The lethal problem was apparently putting a large quantity of thick gel into the venous circulation, where it behaved like injecting countless tiny plugs directly toward the lungs.
Explains it pretty well. Does not explain why he insisted on doing it at all, or why so much at once. This did not increase his wealth.
I would imagine that a careless injection put it into a vein which travels back to the heart, right atrium, right ventricle, lung. Penis has a lot of large vasculature, and I would assume they’re trying to inject all over the place to increase the size.
I don’t know anything about that surgery in particularly, but 40 ml seems like a pretty large volume!
I hope @relaxedmeatball can chime in here, as there are several concerning elements of this diagnosis - perhaps he can explain what the standard process is at this point, is the patient warned, made aware, put on medication, supervised treatment - is the protocol different at the NHS, and therefore by implication he was killed by the negligent care from US providers. How is it handled in Britain? I assume a catherization procedure is mandatory with a 90% aortic stenosis, but this is presumably in an emergency setting and not just being put on a list “we’ll get to you when we can, hold on”. Back in the day this would lead to stenting, but that’s been deprecated, I think LLT and anticoagulants now? Please enlighten us!
He was only 54. Was the “markedly enlarged heart” subsequent to the hypertension diagnosis or idiopathic HCM? Or was this simply because of aortic stenosis? What about ejection fraction? Preserved? Perhaps you can throw some light here.
Nicholas Brendon’s cause of death revealed
https://people.com/nicholas-brendon-probable-cause-of-death-11931448
"A cause of death for Nicholas Brendon has been disclosed by the Putnam County Coroner’s office.
On May 5, coroner Todd Zeiner announced in a report obtained by PEOPLE that Brendon’s death was natural and he died of atherosclerotic and hypertensive cardiovascutar disease. The report lists acute pneumonia and previous myocardial infarction as contributing factors. The “mechanism of death” was 90% blockage right coronary artery."
“Zeiner said a longtime friend of Brendon’s, Theresa Fortier, was at his residence and called 911. “Theresa stated the decedent had been having a persistent cough and had been self medicating with over the counter medications. He had been complaining about chest pain. She recommended a hospital, but he declined,” Zeiner wrote. “He was a longtime smoker and was apprehensive about going due to his recent back surgery. She advised he had a recent heart attack several years ago, but he declined any further treatment at that time.””
“After finding no further evidence at the scene, his body was transported to the coroner for an autopsy, which determined the actor had a “markedly enlarged heart, severe stenosis of the right coronary artery, moderate stenosis of the left anterior descending and left circumflex arteries, acute pneumonia and inflammation of the small bowel. The small bowel inflammation is most likely secondary to ischemic changes related to the cardiogenic shock.””
"
Unfortunately I am not a cardiologist (professor and researcher in the cardiovascular field, but not a clinician) so I don’t think I can make any judgement here about the quality of care, sorry!
My lay person interpretation is that he’s a guy who smoked for a long time, clearly didn’t like doctors and hospitals, and had very advanced cardiovascular disease - with advanced stenosis in all 3 major coronary arteries. He also had a non-fatal heart attack back in 2022. So a death at age 52 isn’t terribly surprising.
The “enlarged heart” is not clearly described. This could be ventricular hypertrophy (i.e. muscle gets larger, often from having to fight against high blood pressure, or abuse of anabolic steroids), or it can be dilated cardiomyopathy, which can happen after injury - such as that prior heart attack in 2022. In the latter, the area of heart muscle which dies doesn’t regenerate, and forms scar tissue (which obviously doesn’t contract), and so the remaining muscle has to work harder. Over time, the entire heart becomes enlarged.
I I had to guess, I’d say the latter. The article says he refused treatment after the 2022 heart attack. Usually, doctors will put you on aggressive blood pressure lowering drugs to reduce cardiac burden.
As for the medical care, to my knowledge, unless you’re unconscious and being wheeled into the OR, you can always decline treatment. The article explicitly says “he had a recent heart attack several years ago, but he declined any further treatment at that time” and that’s his choice to make in the UK, and I’m sure it is in the US also.
Thanks, @relaxedmeatball - I always want to hear from somebody who studies these issues, as there is just so much misinformation out there. I think you are right - he was not a particularly compliant patient. Too bad.
Presley Gerber age 27. Model. Possible drug overdose while in rehab.
He was the son of model Cindy Crawford.
George Chuvalo age 89. Boxer. Complications related to late-life dementia.
Chuvalo had 4 family members (3 sons, and his wife) die from drug overdoses.