This is really interesting feedback, so I’m glad you shared this.
Did you send your CCTA results to clearly yourself? Or your doc did? I’m just trying to figure out what the options are… the two step process vs going to a Cleerly approved center (few and far between).
This makes me wonder if one goes to a place that automatically uses Cleerly, is there a human who also looks at the results? If not, the two step process, as you did, seems more advantageous because you’d get what Cleerly has to offer, AND you would also get what Cleerly might miss, as happened in your case.
I live in New Zealand, so had a normal CCTA done here (with humans). For the Cleerly analysis I had a Cleerly-aligned USA-based Doctor who arranged the Cleerly referral and billing. Once that was complete, my NZ radiology provider transferred my CCTA data files directly to Cleerly (this process was smooth for me and only entailed sending an email to authorise the transfer).
To clarify, normally the people who do the CCTA have a guy that looks at it and gives his opinion. Then the images are sent to Cleerly (there are others with AI too, but this is the one we’re talking about) and they add their opinion.
No, I’ve emailed them and gotten the particulars. I’m waiting for my medicare card, then I’ll talk to them about that and get it done. I’ll post here if it happens.
I did it (CCTA w/Cleerly) recently. I was interested in seeing the status of OM2 (among others) which about 15 years ago was deemed 99% plugged!! This time it was clean as a whistle! There was plenty of plaque, distributed with the bulk being hard plaque, a spot or two around 70% blocked. Since my LDL at below 20 and APOB around 37 are at levels where presumably new plaque will not be deposited I am not perturbed. I am going to resume Nattokinase, Serrapeptase and Lumbrokinase after the New Years, have been on them for over a year. At 67 I have to expect issues. Testing gives me visibility on actions to take and effects of prior actions!
I finally got a blood test and wanted to share the results, which are looking quite good. Unfortunately I don’t have any values from before I started self administering drugs.
Stack prior to the test:
Pitavastatin: 2mg
Ezetimibe: 5mg
Bempedoic Acid: 90mg
Aspirin: 81mg
Metformin: 1g
Rapamycin: 5mg (once a week)
I wonder for how long have you been on this stack (before doing the blood work) and did you also check the fasted glucose? Your stack (which resembles mine, except Bempedoic acid) is great for lipids but not necessarily for glucose (hopefully metformin was enough to keep it in check). In my limited way of understanding health and longevity, lipids kept low is good to stay/keep one alive, glucose kept low is good for health and longevity.
If I were to oversimplify it even further, I’d say managing lipids gets you to 90, whereas also managing glucose gets you another 10-15 years on top of the 90 (total 100-105) and then exercise and 20% calorie restriction is good for another 5-10 and all of the sudden you are at 115-120. Add RAPA, acarbose, and SGLT2 for another 10-15 and now you’re cruising to 140
It was adjusted over time (eg changing from Rosuvastatin to Pitavastatin and adding metformin) but I think it was pretty consistent for at least 2 months before the test.
My glucose levels did seem a bit high alright. Also I appear to be slightly anaemic. I’m wondering what the consensus is for longevity in regards to it.
Other stuff:
blood glucose 87mg/dl.
Hba1c 5.4%.
Red blood cell count: 4.63 million/mcl.
Haemoglobin 13.2 g/dl.
Blood pressure 99/55.
BMI 21.
Age 38.
Male.
AST 26 U/L
ALT 19 U/L
|gamma-GT 11 U/L
Time to start an SGLT2i and acarbose perhaps (if you’re not taking currently). Can’t help with the anemic part, but I’m sure there is others with more knowledge (that can chime in).
Iron-deficiency anemia has pros and cons, but if below the lower lab threshold, the consequences can be deleterious, like fatigue, immune deficiency, and other impaired biological mechanisms. The optimum is probably in the lower quartile or quintile of the distribution. No fatigue. Lowered risk of Fe-driven oxidative signals and consequent increased cancer hazard. a typical U-curve hazard model.
I recently exhibited a slight iron-deficiency, the most immediate symptom being excessive fatigue and somnolence. I am taking Iron bysglicinate supplements. Fatigue ceased. I still have to repeat the labs.
I took it because the consensus is that chelated Iron, bisglycinate in this case, has some advantages, usually, but not in all cases. The main disadvantage is the higher cost. But not too much. All in all, it seems to be the optimal choice. And absorption is usually improved if taken every other day. This procedure seemingly inhibits the hepcidin anti-absorptive action.
A heme iron supplement such as Proferrin has major potential advantages over all the others including bisglycinate, chief of which is the convenience of being able to take it with food and likely better absorption and less risk of side effects. It worked extremely well for me.
